Mitochondrial Activity in Endothelial Cells

This article discusses the fact that endothelial cells apparently make 75 percent of their ATP from glycolysis and are not supposed to be highly oxidative [Sean Davidson and Michael Duchen, 2007: (http://circres.ahajournals.org/cgi/content/full/100/8/1128) (http://www.ncbi.nlm.nih.gov/pubmed/17463328)]. The assumption that reliance on glycolysis equates with independence of mitochondrial activity seems questionable to me, given that mitochondrial activity can help maintain glycolysis in the cytosol. But I guess that's what the authors are getting at, and they say that endothelial cell mitochondrial functioning has been neglected. They discuss research on the sensitivity of coronary endothelial cells to hypoxia. The authors imply that the resistance of cultured endothelial cells to hypoxia may have caused researchers to think of endothelial cells as being able to easily maintain ATP levels during hypoxia, and this seems not to be the case.