Ethanol and Nucleotide-Derived Ribose-5-Phosphate and Thiamine Turnover

I was reading about thiamine partly because ethanol intake has been associated (for a long time) with thiamine depletion (increases in the rate of turnover of thiamine cofactors, I guess one would say). Ethanol increases the cytoplasmic NADH/NAD+ ratio and lactate/pyruvate ratios (http://jasn.asnjournals.org/cgi/content/full/12/suppl_1/S15 pubmed (Friedrich Luft): http://www.ncbi.nlm.nih.gov/pubmed/11251027?dopt=Abstract), and that's similar to the effect that xylitol or ribose-5-phosphate, derived from uridine or purine nucleotides, can have (http://hardcorephysiologyfun.blogspot.com/2008/12/fructose-at-least-its-good-for.html and http://hardcorephysiologyfun.blogspot.com/2008/12/reducing-sugars.html). There wouldn't be all that much ribose-5-phosphate (derived from ribose-1-phosphate) from nucleotides, but I'm thinking it might have an effect of increasing thiamine turnover. There must be a lot of research on the mechanistic interrelationships of ethanol and thiamine, but I haven't read much on that.