I was going to clarify something about the type of magnesium-induced neuromuscular effect that is described in this type of article:
http://www.ncbi.nlm.nih.gov/pubmed/8652332
This isn't caused by magnesium per se but by the fact that magnesium is reducing the release of ACh in the face of the already-blunted neuromuscular transmission, the effect of residual amounts of the neuromuscular blocking agent used during the operation. The magnesium potentiates the effect of the remaining amount of the nicotinic acetylcholine receptor antagonist (vecuronium, in one of those articles) (and in the reports that show actual recurarization in patients):
http://www.ncbi.nlm.nih.gov/pubmed/7734259
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